Western medicine postulates: “Neuromuscular disorders affect the nerves that control voluntary muscles and the nerves that communicate sensory information back to the brain. Nerve cells (neurons) send and receive electrical messages to and from the brain to help control voluntary muscles. When the neurons become unhealthy or die, communication between the nervous system and muscles breaks down. As a result, muscles weaken and waste away (atrophy).” Is this true?
SYMPTOMS: These neuromuscular disorders result in muscle weakness and fatigue that progress over time. Some neuromuscular disorders have symptoms that begin in infancy, while others may appear in childhood or adulthood. Symptoms will depend on the type of neuromuscular disorder and the areas of the body that are affected. Some common symptoms of neuromuscular disorders include muscle weakness that can lead to twitching, cramps, aches and pains, muscle loss (atrophy), movement issues, balance problems, numbness, tingling or painful sensations, droopy eyelids, double vision, trouble swallowing, trouble breathing.
CONDITIONS OF NEUROMUSCULAR DISORDERS INCLUDE Amyotrophic lateral sclerosis (ALS), Charcot-Marie-Tooth disease, Multiple sclerosis (MS), Muscular dystrophy, Myasthenia gravis, Myopathy, Myositis, including polymyositis and dermatomyositis, Peripheral neuropathy, Spinal muscular atrophy.
DIAGNOSIS: Checking a patient’s reflexes and muscle strength, as well as evaluating other symptoms, may lead a physician to order other diagnostic tests, including blood test to check for elevated enzymes, magnetic resonance imaging (MRI) scan of the brain and spinal cord, lumbar puncture (spinal tap) to check the cerebrospinal fluid, electromyography (EMG) to record the electrical activity of each muscle, nerve conduction studies to see how well signals travel from nerve to muscle, muscle biopsy to examine a sample of muscle tissue under a microscope, genetic testing to confirm gene mutations. Medicine considers all above-mentioned neuromuscular conditions as not treatable.
We have in our body muscles striated (voluntary) usually related to the skeletal body and smooth muscles (involuntary) in charge of peristaltic and other ANS controlled functions.
Neuromuscular disorders are affecting both groups of muscles. Therefore an MS patient first will lose its mobility, and as the progress of the condition, one may lose breathing reflexes.
The striated muscles developed at a time when more efficient muscles’ functions were required. The striated muscles of the skeletal musculature, myocardium ventricles, coronary arteries, coronary veins, aorta, carotid arteries, and subclavian arteries, blood vessels, tongue, jaw, ear, bronchi, larynx, diaphragm, esophagus, stomach small curvature, pylorus, duodenal bulb, pancreatic ducts, bile ducts, gallbladder, cervix, cervical sphincter, vagina, rectum, external rectal sphincter, renal pelvis, ureters, urethra, bladder, external bladder sphincter, eyelid muscles ciliary muscles, and extraocular muscles derive from the NEW MESODERM.
The CEREBRAL MEDULLA controls the trophic (nutritional) function of the striated muscles. The MOTOR CORTEX controls the ability to move the muscles.
In a biological conflict (stressor), the related muscles generate during the conflict-active phase cell loss and muscle paralysis. In the healing phase, the muscles are reconstructed. There may be muscle cramps, rhythmic convulsions, spasms, or muscle twitching during the healing phase.
DEVELOPMENT AND FUNCTION OF THE SKELETAL MUSCLES: The musculoskeletal system provides the form to the body and allows the body to move and maintain its posture. The muscles are connected to the bones and joints through tendons and ligaments and are endowed with connective tissue, nerves, and blood vessels. The skeletal muscles are composed of bundles of fibers organized in a striped pattern; therefore, they are called striated muscles. Skeletal muscles vary considerably in shape and size. They range from extremely tiny strands such as the middle ear’s stapedius muscle to large masses like the thighs’ muscle.
BRAIN LEVEL: The skeletal muscles have two control centers in the cerebrum. THE TROPHIC function of the muscle, responsible for the muscular tissue’s nutrition, is controlled from the CEREBRAL MEDULLA; THE CONTRACTION of the muscles is controlled from the MOTOR CORTEX (part of the cerebral cortex).
The muscles of the right side of the body are controlled from the cerebrum’s left side; the left side muscles are controlled from the right cerebral hemisphere. Hence, there is a cross-over correlation from the brain to the organ.
In the cerebral medulla, the bones, skeletal muscles, lymph vessels with lymph nodes, blood vessels, connective tissue, and fat tissue share the same brain relays and, therefore, the same biological conflict (stressor), namely a SELF-DEVALUATION CONFLICT. The control centers are orderly positioned from head to toe.
The biological conflict linked to skeletal muscle atrophy is a moderate self-devaluation conflict. The specific self-devaluation conflicts are the same as for the bones and joints. In line with evolutionary reasoning, self-devaluation conflicts are the primary conflict associated with cerebral medulla-controlled organs deriving from the new mesoderm.
The biological conflict (stressor) related to the movement impairment and paralysis of the muscles is a MOTOR CONFLICT of “not being able to move” or “feeling stuck.” The conflict can be associated with the entire body (generalized motor conflict) or with a single muscle or muscle group (localized motor conflict).
In this case, the biological conflict (psychogenic triggers or stressor) is different based on the major function of that part of the body:
FACIAL MUSCLES: losing face, loss of status, reputation, respect, honor, prestige, dignity; disgrace, humiliation, shame, being exposed, feeling ridiculed, foolish or stupid
JAW MUSCLES: not being able to bite
NECK MUSCLES: not being able or allowed to move or turn the head
SHOULDER AND BACK MUSCLES: not being able to get out of the way or step aside
ARM MUSCLES: being forcefully held down (physical abuse, sexual abuse, during a vaccination, in a fight or “play”), not being able to hold or embrace someone or hold someone back (flexor muscle), not being able to push someone away, fight somebody off, or defend oneself (extensor muscle and muscles around the elbows)
HAND MUSCLES: not being able to hold on to someone or hold someone back (a loved one who is leaving or dying); not being able to grab something; any distress associated with the hands (work, hobby, or sports-related)
LEG MUSCLES: not being able to escape, flee, or run away (literally or figuratively, e.g., from a workplace or a relationship), not being able to leap aside, not being able to follow, feeling rooted to the spot (petrified), feeling trapped (literally or figuratively), not being able to keep up, not being able to climb up (e.g., not being promoted), not being able to kick somebody away (extensor muscle), a fear of not being able to walk (wheelchair image).
Motor conflicts can also be experienced with or on behalf of someone else, particularly when “feeling stuck” concerns a loved one.
The belief that conditions such as ALS or MS are hereditary makes a family member more susceptible to conflicts of the same kind. A fetus might endure the conflict of “not being able to escape” when the mother is in danger or because of threatening noises in the immediate environment (jackhammers, chainsaws, lawnmowers, grass trimmers), loud kitchen equipment such as blenders held close to the womb, or screaming and yelling (fights between parents, mother yelling at her children). In this case, the baby is born with (partial) paralysis of the legs and motor disabilities if the conflict is not resolved. The “loud noises” of ultrasound examinations can be highly traumatic for the unborn (see Down syndrome). A “feeling stuck” conflict could be activated during a difficult delivery or how the baby is handled immediately after birth.
During the CONFLICT-ACTIVE PHASE, there is a cell loss (necrosis) of muscle tissue (controlled from the cerebral medulla) and, at the same time, muscle weakness or muscle paralysis (controlled from the motor cortex). With the impact of the motor cortex’s conflict, fewer nerve impulses are transmitted to the corresponding muscle, causing a loss of muscle function (compare with sensory paralysis related to the epidermis and the periosteum). The muscle weakness might be noticed as clumsiness or heaviness when the legs are affected.
Prolonged conflict activity leads to muscle atrophy (WEI SYNDROME) without paralysis if the conflict is experienced solely as a self-devaluation conflict. The pelvic floor muscles become weak because of a difficult pregnancy, sexual humiliation, chronic constipation, or urinary incontinence, making them feel “worthless” there.
If additionally, a motor conflict occurs, muscle atrophy and muscle paralysis manifest together, particularly when the distress of not moving an arm or leg (or both) causes a self-devaluation conflict.
Muscle weakness and muscle paralysis were formerly diagnosed as paralytic poliomyelitis, or “POLIO,” purportedly a “viral infection” that mainly affects children (the scientific evidence of the existence of a “poliovirus” has never been provided!). In the Western World, where polio was supposed to be eradicated by vaccination, the same ALS symptoms (Amyotrophic Lateral Sclerosis, also known as Lou Gehrig’s disease or motor neuron disease), MULTIPLE SCLEROSIS, or Guillain-Barré syndrome.
As presented in Parkinson’s and Huntington’s disease, movement disorders are considered inherited “neurodegenerative diseases.” This brain CT on the right shows the impact of a motor conflict. The focus’s center is on the right brain hemisphere (para-central), precisely, in the motor cortex that controls the left leg. The partly edematous ring (dark) indicates that the healing phase is interrupted by conflict relapses (sharp borders), hence the legs’ continued weakness, predominantly of the left leg.
Whether the muscle atrophy or muscle paralysis occurs on the right or left side of the body (or on both sides) is determined by a person’s handedness and whether the conflict is mother/child or partner-related. A localized conflict affects the muscle or muscle groups that are associated with self-devaluation or motor conflict.
Muscle(s) weakness (WEI syndrome) and a loss of sensitivity in the feet, legs, or arms are considered as one of the first symptoms of multiple sclerosis (MS). The big danger is that the patient suffers a motor conflict due to the diagnosis shock since he has been told that he will most likely be in a wheelchair for the rest of his life.” MS diagnosis causes a great panic, affecting at later stages kidneys and lungs.
The same holds when a person is diagnosed with ALS. The fear of not being able to walk and ending up in a wheelchair (“feeling stuck”) is so overwhelming that the motor conflict which had given rise to the first symptoms often becomes irrelevant. With the paralysis progression, the muscle atrophy also advances, leading to clumsiness, difficulties walking, and frequent falls (see “falling conflict” and vertigo). This activates the additional motor and self-devaluation conflicts because the mobility becomes more and more impaired, and the prognosis becomes a self-fulfilling prophecy.
The belief that MS, ALS, or Parkinson’s are “hereditary diseases” makes a person whose parent has the condition more vulnerable to suffer a motor conflict (conflicts can also be experienced on behalf of someone else). The subsequent symptoms lead quickly to the same diagnosis, genetic hereditary condition!!
In conventional medicine, it is assumed that multiple sclerosis is caused by a “degradation of the myelin sheath” concluded from MRI brain images. The myelin sheath is an insulating layer that envelops nerves, including nerves in the brain and spinal cord. The “myelin destruction” is thought to be an “autoimmune response” where the immune system “mistakenly” destroys the myelin sheath covering the motor neurons in the brain.
As the immune system theory, the concept of “autoimmune disorders” that supposedly damage healthy body tissue is an academic construct that has no scientific basis. The claim that MS is the result of a “destruction” of the myelin sheath is highly questionable. On the MRI, the suspected “demyelination” (called “MS plaque”) shows in the cerebral medulla, specifically, in the area that controls the muscles (trophic function) around the right hip (self-devaluation).
Neurologists consider “the abnormal white area” as the reason for the paralysis. The “MS plaque” accumulates neuroglia, indicating that the person is trying to heal a self-devaluation conflict caused by the motor paralysis (controlled from the motor cortex). NOTE: The myelin sheath is controlled from the cerebellum and linked to a touch conflict. If a build-up of neuroglia is found in the motor cortex, then the “MS plaque” is diagnosed as a “brain tumor,” usually followed by excision of the lesion (see also “brain tumor seizures”).
In Eastern medicine (TCM) the muscle atrophy and weakness are known as WEI syndrome. WEI Syndrome refers thus to a group of disorders with symptoms ranging from flaccidity and weakness in the sinews and muscles to diminished muscle mass. Muscular atrophy may be localized or systemic. The lower extremities are often more affected, resulting in weakness and difficulty ambulating. As the voluntary movement’s capacity diminishes, unremitting Wei Syndrome progresses to a total loss of mobility of knees, hips, shoulders, arms, or spine. WEI syndrome affects the muscle’s trophic function, is therefore caused by a self-devaluation conflict.
NOTE: In TCM, WEI syndrome appears in the general symptomatic picture of specific deficiencies (Qi, Yin, Yang, Jing) affecting different organs such as lungs, spleen, stomach, kidneys. Therefore, the treatment of WEI syndrome is at the same time therapy for different conditions affecting those organs.
SYMPTOMS: In western medicine, WEI syndrome is associated with sequelae of encephalitis, spinal cord trauma, muscular atrophy due to polio, multiple sclerosis (MS) tumors the CNS. It shows in many diseases, such as muscular atrophy, muscular dystrophies, myotonic syndromes, periodic paralysis, myasthenia gravis, endocrine myopathies, hysterical paralysis, etc. There is no cure for all the above-mentioned conditions,
At least on the western medicine side.
In TCM, WEI syndrome or Atrophy syndrome is considered to be caused by injuries to the internal organs, especially insufficient Blood and Yin Deficiency with excessive Fire; therefore, symptoms of Heat or Deficiency are common.
- Malnourishment of channels and tendons due to exhaustion of body fluid invokes exogenous pathogenic wind-heat to the lungs. The lung is the organ that faces all channels and collaterals and keeps the normal circulation of Qi and blood (in TCM theory)
- Obstruction and lesion of the channels and tendons due to accumulation of damp-heat caused by living in a damp area; or eating fat, hot and spicy food; or drinking too much alcohol
- Malnutrition of channels and tendons due to the Qi and blood deficiency which was caused by the dysfunction of the spleen and stomach
- Malnutrition of channels and tendons due to the insufficiency of the liver and kidney essence and Qi in the later stage of chronic diseases.